Markets Gain In the operating room revealed a 5.3% incidence of arterial hypoxia, w During hypercapnia in 27.3% of them observed. None of these problems developed respiratory depression requiring intubation. In sp Th awakening pts, the incidence of arterial hypoxia 18%, w While the Danusertib Aurora Kinase inhibitor H FREQUENCY of hypercapnia was 8.3%. The analysis of kardiovaskul Ren no complications developed in group pt waking early postoperative administration were hypotension, vasopressors. In the sp Th awakening group, 4.3% of the values hypotensive pt (2.9%, the use of vasopressors. In the early stimulation group, the incidence of hypertension (antihypertensive medication required was 14.3%, w short while for the term postoperative sedation points, the incidence of hypertension (using antihypertensive drugs was 21.5%.
We observed no difference in length of stay postoperatively in the ICU between the two groups, no differences XL147 956958-53-5 in the H FREQUENCY of occurrence severe neurological complications (postoperative hemorrhaghe or brain that. CONCLUSION. Pts allowed to awaken early after brain tumor surgery incidence was increased ht by hypercapnia, w while points to a short-term postoperative sedation seems anf to be with propofol lliger for arterial hypox chemistry and mainly exposed to high blood pressure. Therefore, ben term, the two M possibilities anesthesia therapy meshed monitoring and h hemodynamic disorders of the respiratory tract (and m aligned therapies of the early morning hours of admission to the ICU. 0669 increased hte serum levels of S100B protein interleukin-6 in patients with h RELEVANT hemorrhagic shock Stathopoulos1 A.
, CI Routsi1, E. Garini1, K. Glynos1, C. Psachoulia2, p Nanas1, C. Roussos1, E. Stamataki3 1ICU, 2Biochemistry , 3Anesthesiology, H Pital Evangelismos, Athens, Greece INTRODUCTION. experimental h hemorrhagic shock induces increased hte production of the protein S100B, a marker of brain injury associated with the severity of shock (1, exclusively whether this increase Lich cerebral hypoperfusion or other factors also contribute to this increase remains unclear. inflammatory response after acute hemorrhage as well (2, and k nnte have an r in the release of S100B. We examined the relationship between serum protein S100B and IL -6, and their meters adjusted prognostic value in patients with h hemorrhagic shock. METHODS.
Eighteen (12 m nnliche patients with a mean age of 6222 yr, undergoing emergency surgery for contr lh hemorrhagic shock different etiology were included. None suffered before Hirnsch the other neurological diseases. Blood samples were taken for measurements of blood gases, lactate, IL-6 and S100B at admission and may need during the n next three days. Vital signs were recorded and the severity of the disease was APACHE II score assessed. RESULTS. The output values of serum S100B protein were obtained ht (median 0.46 lg / l, IQR were 3.5 and 0.16 positively correlated with IL-6 (R0.8, p \ 0.01, lactate (r0.63, p \ 0.01, and the result h Pital (R0.5, p \ 0.05. For all data, there was a significant correlation between the levels of S100B and IL-6 (r0.46, p \ 0.01, lactate (r0.49, p \ .01 APACHE II score (r0.38, p \ 0.
01 and results (r0.54 was p \ 0.01. mortality 44%. serum S100B and IL-6 levels at admission was significantly h ago in patients who died compared to survivors (3.7 vs. 3.6 0.71.05 lg / l, p \ 0.01 and 505 was 409 against 138 197 pg / ml, p \ 0.01, respectively. In the linear regression analysis APACHE II score is a independent ngiger Pr predictor of outcome (F13.9; p \ 0.01. CONCLUSION. serum S100B protein in patients with increased Hten h hemorrhagic shock, and this increase with the IL-6, the severity of disease and lactate results correlated. These results show that, stimulates resembled with the exception of cerebral hypoperfusion m, the release of S100B by be may be other factors that lead to inflammatory response and / or adversely chtigten tissue perfusion. REFERENCE (S.
1 Shock, 2003, 19:422, 2nd Ann Surg 1993, 218:769. recognition weight. in part by the Foundation found rderten thorax. 0670 survey on the use of external ventricular drains ren (DEA AND DRAINAGE cerebrospinal fluid (CSF Sch del Brain Injury (TBI Reddy1 K., M. Healy2, V. Verma2 1ICM, Universit tsklinikum Lewisham , 2ICM, the Royal London Hospital, London, UK INTRODUCTION prime. That re aim of this study was to evaluate the current practice of using DEA in intracranial pressure (ICP and CSF drainage increased to Hten ICP monitoring in TBI METHODS deal with all neuroscience centers Britain in Wholesalers. electronic questionnaires Gen. were sent to lead clinicians in 35 adult and p pediatric intensive care units of neurosurgery and neuroscience. A copy of the survey carried out to all non-responders were sent. goal was to provide data about the type of ICP monitor use, the H FREQUENCY using EVD and CSF drainage, including normal indications, indications and complications of the resistance, collect associated with their use. RESULTS. Forty-eight completed questionnaires have come into effect, the 31 neurosurgery and intensive care units. or the